Fwd: New laws to help Vic asbestos victims (AAP via Yahoo!7 News)

---------- Forwarded message ----------
From: Yahoo! News Search Results for asbestos cancer <rssfwd@rssfwd.com>
Date: Fri, May 23, 2008 at 7:59 PM
Subject: New laws to help Vic asbestos victims (AAP via Yahoo!7 News)
To: mesothelioma77@gmail.com


Asbestos victims in Victoria will have greater power to seek compensation as the state government moves to end an agonising legal choice they now face.

Fri, 23 May 2008 07:50:13 GMT

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Source: http://us.rd.yahoo.com/dailynews/rss/search/asbestos+cancer/SIG=11gativk5/*http%3A//au.news.yahoo.com/080523/2/16zd0.html
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Fwd: Primary peritoneal mesotheliomas in children: a clinicopathological and immunohistochemical study of eight cases.

---------- Forwarded message ----------
From: HubMed - mesothelioma cancer <rssfwd@rssfwd.com>
Date: Fri, May 23, 2008 at 7:59 PM
Subject: Primary peritoneal mesotheliomas in children: a clinicopathological and immunohistochemical study of eight cases.
To: mesothelioma77@gmail.com


[1]Histopathology. 2008 Jun; 52(7): 824-30
Moran CA, Albores-Saavedra J, Suster S

AIMS: To present eight cases of primary diffuse peritoneal malignant mesothelioma in children

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Source: http://www.hubmed.org/display.cgi?uids=18494612
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Fwd: Nanotubes may be as bad as asbestos (Electronics Supply and Manufacturing)

---------- Forwarded message ----------
From: Yahoo! News Search Results for asbestos cancer <rssfwd@rssfwd.com>
Date: Fri, May 23, 2008 at 7:59 PM
Subject: Nanotubes may be as bad as asbestos (Electronics Supply and Manufacturing)
To: mesothelioma77@gmail.com


PORTLAND, Ore. — Carbon nanotubes could cause the same maladies as asbestos, according to a study by University of Edinburgh, which also showed that long-thin fibers cause the pathological response known to be a precursor to mesothelioma cancer in mice.

Fri, 23 May 2008 22:52:13 GMT

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Source: http://us.rd.yahoo.com/dailynews/rss/search/asbestos+cancer/SIG=126l9dvrt/*http%3A//www.my-esm.com/digest/showArticle.jhtml?articleID=208200378
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Fwd: Multiple roles of nicotine on cell proliferation and inhibition of apoptosis: Implications on lung carcinogenesis.

---------- Forwarded message ----------
From: HubMed - mesothelioma cancer <rssfwd@rssfwd.com>
Date: Fri, May 23, 2008 at 7:59 PM
Subject: Multiple roles of nicotine on cell proliferation and inhibition of apoptosis: Implications on lung carcinogenesis.
To: mesothelioma77@gmail.com


[1]Mutat Res. 2008 Apr 11;
Catassi A, Servent D, Paleari L, Cesario A, Russo P

The genotoxic effects of tobacco carcinogens have long been recognized, the contribution of tobacco components to cancerogenesis by cell surface receptor signaling is relatively unexplored. Nicotine, the principal tobacco alkaloid, acts through nicotinic acetylcholine receptor (nAChR). nAChR are functionally present on human lung airway epithelial cells, on lung carcinoma [SCLC and NSCLC] and on mesothelioma and build a part of an autocrine-proliferative network that facilitates the growth of neoplastic cells. Different nAChR subunit gene expression patterns are expressed between NSCLC from smokers and non-smokers. Although there is no evidence that nicotine itself could induce cancer, different studies established that nicotine promotes in vivo the growth of cancer cells and the proliferation of endothelial cells suggesting that nicotine might contribute to the progression of tumors already initiated. These observations led to the hypothesis that nicotine might be playing a direct role in the promotion and progression of human lung cancers. Here, we briefly overview the role and the effects of nicotine on pulmonary cell growth and physiology and its feasible implications in lung carcinogenesis.



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Source: http://www.hubmed.org/display.cgi?uids=18495523
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